A new peer-reviewed study has quietly revealed one of the most consequential biological findings of the pandemic era — and the authors never acknowledge it: Every single vaccinated participant in the study had fibrinolysis-resistant, ThT-positive amyloid microclots circulating in their blood.
Hidden in the supplementary tables is a demographic and biochemical pattern that completely reframes the paper:
94% of all participants were vaccinated.
100% of these vaccinated individuals had amyloid microclots — including every “healthy control.”
The condition labeled “Long COVID” occurred almost entirely in a heavily vaccinated population, without any laboratory confirmation of prior SARS-CoV-2 infection. In reality, the study is observing Long VACCINE pathology, not Long COVID.
And because the authors’ own mechanistic experiments show that purified spike protein alone produces these amyloid, fibrinolysis-resistant clots, the implications are profound.
All individuals in the study — 100% of the vaccinated — had amyloid microclots
Researchers identified microclots using Thioflavin-T (ThT), an amyloid-binding fluorogenic dye. ThT positivity was the defining criterion. A structure was only counted as a microclot if it bound ThT.
Therefore, every microclot counted in the study is, by definition, amyloidogenic.
And according to Table S11, every single vaccinated participant had amyloid microclots in multiple size ranges:
Because 83 of 88 participants (94%) were vaccinated, this means:
Every vaccinated person in the study had amyloid microclots.
“Long COVID” (Long VACCINE) patients had extreme elevations in large, pathological amyloid microclots
Small amyloid microclots were present in everyone, but the pathological burden differed sharply.
According to Table S11:
98% of “Long COVID” (Long VACCINE) patients had large microclots in the 900–1600 µm² range
60% had very large microclots >1600 µm²
Total microclot burden was ~20-fold higher in “Long COVID” patients
These larger, pathogenic amyloid microclots were densely packed with:
Neutrophil extracellular traps (NETs)
Myeloperoxidase
Neutrophil elastase
Extracellular DNA
Misfolded amyloid fibrin
COVID-19 infection was never verified
Despite positioning the results as a hallmark of “Long COVID,” none of the participants were confirmed to have had SARS-CoV-2 infection. The study performed:
no antibody testing
no PCR
no sequencing
no neutralizing antibody assays
Long COVID status was assigned purely via symptoms and clinician impression. There is no evidence in the study that any participant was biologically positive for prior infection.
Thus, the clotting abnormalities cannot be attributed specifically to infection, but rather to vaccination.
Spike protein alone produced identical amyloid microclots
In a mechanistic experiment, the authors added purified spike protein to fibrinogen.
This single intervention produced:
insoluble, ThT-positive amyloid microclots
misfolded fibrin structures identical to those in patient samples
fibrinolysis-resistant aggregates compatible with vessel obstruction
The authors confirmed that Spike protein directly induces amyloid microclot formation, corroborating previous studies.
Explains prevalent white fibrous clots found in the dead
The study’s core findings — 100% amyloid microclots in vaccinated individuals and direct spike-induced amyloid fibrin formation — offer a clear mechanism for the large, rubbery white fibrous clots increasingly reported in deceased individuals since 2021.
At the 2025 Tennessee Funeral Directors Association (TFDA) convention, former USAF Major Tom Haviland conducted the first state-level survey of embalmers:
64% reported white fibrous clots in 2025
Found in 17% of all bodies
70% observed widespread microclotting (“coffee-grounds blood”)
39% reported rising infant deaths (+14%)
BREAKING: Tennessee Funeral Directors Association Confirms White Fibrous Clots Are Real and Prevalent
·
16 jun
By Nicolas Hulscher, MPH
Forensic analysis by Kevin W. McCairn, PhD et al shows that these postmortem clots:
are amyloidogenic fibrin aggregates, not normal thrombi
exhibit β-sheet structures (ThT-positive)
are protease-resistant, rubbery, and fibrous
have dense fibrillar ultrastructure on SEM
contain human genetic material
and show preliminary plasmid/spike-associated markers
These characteristics match exactly the pathological microclots described in the new study — only at a later, aggregated, end-stage form.
The progression is biologically straightforward:
Spike exposure (infection or mRNA vaccination)
Amyloid microclots form — present in 100% of vaccinated subjects
Large, NET-rich, fibrinolysis-resistant clots accumulate (20× higher in Long vaccine patients)
These merge into massive, rubbery, white fibrous intravascular clots
This new study documents the early and intermediate stages in the living; Haviland’s surveys and McCairn’s analysis reveals the final stage in the dead.
Conclusions
Although the authors frame their findings as “Long COVID,” the underlying data reveal something far more consequential:
100% of vaccinated participants had amyloid microclots.
Large, fibrinolysis-resistant amyloid microclots were concentrated in the Long vaccine group.
No participant had laboratory-confirmed SARS-CoV-2 infection.
Spike protein alone produced identical amyloid microclots in vitro.
With 94% vaccination uptake, the biological signal is overwhelmingly linked to spike exposure in a vaccinated population.
These findings carry serious public-health implications:
Every vaccinated individual in the study showed early-stage amyloid microclots, raising alarms about cumulative vascular injury across the entire globe.
The pathology mirrors the large white fibrous clots now documented by embalmers and forensic analysts.
And critically:
The CDC and federal public-health agencies must finally do their job and launch an immediate, transparent investigation into these findings.
Failing to intestigate the white fibrous clot situation constitutes a dereliction of duty.
Any platform delivering spike protein into human circulation must be immediately banned for human use.
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